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There are several possible reasons: Oral absorption challenges Variable bioavailability Differences in formulation technology Inconsistent potency between products Many patients report weaker appetite suppression and less predictable results compared to injections

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Parallel intake of any cardiovascular medications is also not desired

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He Y, Huang Y, Xu H, Yang X, Liu N, Xu Y, et al

GLP-1R activation of neuroprotective pathways in neurons, microglia and astrocytes has also been reported: improvements in overall cognition, learning and motor function potentially associated with GLP-1RA administration in Alzheimers disease and Parkinsons disease may be mediated not only by their amyloid pathology-ameliorating properties (A, tau and -synuclein), but also the suppression of Ca 2+ deregulation and endoplasmic reticulum stress, anti-inflammatory activity, blockage of oxidative stress, mitochondrial dysfunction and apoptosis pathways, enhancements in the neuronal insulin sensitivity and energy metabolism, functional improvements in autophagy and mitophagy, elevated brain-derived neurotrophic factor and glial cell line-derived neurotrophic factor synthesis, and neurogenesis 36