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While cytosolic and blood GSH and Cys biochemical species all experience depletion, the model predicts that a single exposure to doxorubicin only results in a minimal perturbation of the antioxidant defence system
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JAMA, 329(16), 14011401
albicans pathogenicity via nutrient competition and acidic microenvironment maintenance, while microbiota dysbiosis and fungal virulence factor secretion drive fungal overgrowth, vaginal epithelial damage and inflammatory responses that induce VVC
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