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Studies had found that APE1 was overexpressed to repair DNA damage in non-small cell lung cancer (NSCLC) tumor tissues, and patients with low APE1 levels significantly increased progression-free survival and overall survival [25]

However, the larger issue isnt with the effects of the medication, but the cost

Ongoing Phase 3 programs extend these findings by examining long-term metabolic stability and cardiovascular implications of glucagon-inclusive therapies

It should answer the reason for denial directly