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In a conventional view, the ubiquitin activating and proteasomal degradation steps hold most potential since enzymes in each step harbor well-defined activity pockets, although they also suffer from issues of specificity since they can affect hundreds or even thousands proteins that are regulated by the UPS

doi:10.3389/fphar.2024.1439536 Bibliographic records for the above were verified against PubMed

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We show that this mitochondrial plasticity is driven by a need to resolve nucleotide stress via ATP-consuming UCK2

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