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illustrated how the selective permeability of the blood-brain-barrier (BBB) and bloodCSF barrier, combined with the spatial distribution of glutathione-metabolizing enzymes, underlies region-specific vulnerability to oxidative injury in the CNS (9)

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In a rat model of spinal nerve ligation (SNL), exenatide significantly enhanced the expression of M2 microglial markers, including IL-10, IL-4, Arg1, and CD206, in both spinal microglia and primary cultured microglia [63]