Pay in 4 interest-free payments of $6.31 Learn more
Ships within 48 hours · Estimated delivery Aug 27 - Sep 1
Preclinical studies in animal models have reported associations with metabolic and adipose-tissue endpoints
doi: 10.1093/qjmed/hct051 Note: Originally published in February 2015, this article has been revised and updated for accuracy and thoroughness.
The mechanism of AOD 9604 in subcutaneous adipose tissue Activation of fat breakdown (lipolysis) AOD 9604 promotes fat breakdown through the following pathways: Activation of the 3-AR signaling pathway: AOD 9604 binds to 3-AR on the adipocyte membrane, activates adenylic acid cyclase (AC), increases the intracellular cyclic adenosine monophosphate (cAMP) level, and thereby activates protein kinase A (PKA)
In addition, we showed that GSH depletion induces autophagy and SIPS, with autophagy being a negative regulator of SIPS
How Treatment Works Personalized and guided by your provider based on your individual needs
Avoid combining in the same application with strong acids (high-concentration AHA or BHA) because pH extremes can destabilize the copper complex
