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To explore the enhanced dependency on mitochondrial function, we further explored the acute metabolic stress imposed by 5FU treatment

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For patients whose clinical situation requires moderate rather than maximal weight loss, such as those with early-stage metabolic dysfunction where 10-12% weight loss is sufficient to normalize metabolic parameters, orforglipron's efficacy profile may be adequate and its oral convenience advantageous

Thus, roGFP2 oxidation by PfGrx occurs preferentially via a glutathione-dependent monothiol and not a glutathione-independent dithiol mechanism (Fig