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Hyperandrogenism is a fundamental component of the pathophysiology of PCOS and primarily drives various phenotypic features, including follicular dysplasia, impaired ovulation, endometrial disorders, and metabolic dysfunction 20,21,22
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Additional quantification by PRM were performed on closely eluting VVVFIKPTC(GSH)PYC(O3)R and VVVFIKPTC(O3)PYC(GSH)R, when necessary
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