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However, while p53 modulates SLC7A11 expression, the expression and activity of SLC7A11 and GPX4 are not associated with SAT1, and only ferrostatin-1 can inhibit ROS-induced ferroptosis in Tet-on cells (Ou et al., 2016)
Unfortunately, this initial processing creates toxic byproducts called free radicals
PubMed: 12750433 TB-4 wound healing dermal repair acceleration with TB-4 in wound models: Malinda KM et al
In addition, RSV can suppress the occurrence of ferroptosis in the myocardium by increasing SLC7A11/GSH and GPX4 [193, 195]
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