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Its most consistently documented mechanism involves VEGFR2 activation and Akt-eNOS signaling, promoting angiogenesis in connective tissue research models (Hsieh et al., Journal of Molecular Medicine, 2017, DOI: 10.1007/s00109-016-1488-y)
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Antimycin-like interaction of sulforaphane with the mitochondrial respiratory chain at the complex III level generates ROS, leading to membrane lipid peroxidation and 4-hydroxynonenal production (Sharma et al., 2010)
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(Details rates of nausea, vomiting, constipation, diarrhoea and dyspepsia.) PMC Martinez V
2024;7(1):e462
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