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HBx-elevated SIRT2 promotes HBV replication and hepatocarcinogenesis
The oxidation of the dithiol groups of Trx(SH) 2 consequently results in the formation of intra-chain disulfide bridges and hence the oxidized form of Trx proteins (TrxS 2 )
These are supportive benefits, not guaranteed treatment outcomes
Apart from the coding region, variable 5 and 3 noncoding regions (UTR) were identified, resulting in transcript heterogeneity
Side effect profiles under identical monitoring conditions
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