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It is rapidly cleared by renal filtration and degraded by proteases before sustaining meaningful receptor engagement in preclinical in vivo systems

Here's where most protocols fail: never inject air into the peptide vial to 'equalise pressure'

It induces G2/M cell cycle arrest via cyclin-dependent kinases and triggers dose-dependent apoptosis and inhibits histone deacetylase by its metabolites in vitro (13)