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IL-1 and TNF- modulation of proliferated and committed myoblasts: IL-6 and COX-2-derived prostaglandins as key actors in the mechanisms involved

To kluczowy mechanizm ochronny, poniewa stres oksydacyjny osabia komrki, czynic je podatnymi na urazy, przyspiesza procesy starzenia oraz zwiksza ryzyko infekcji

That decline is part of why it's drawn research interest in the first place: if a molecule occurs naturally and its levels drop as tissue repair capacity drops, it becomes a reasonable candidate for studying repair signaling directly

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