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These digest relatively quickly despite GLP-1 slowing, and they provide complete amino acid profiles including the leucine necessary to trigger muscle protein synthesis signaling
1995;173(3 Pt 1):7607
Circulation 146, 18951897
394 Mutations in C10orf2 disrupt mtDNA maintenance, leading to secondary mtDNA depletion and subsequent mitochondrial dysfunction
This difference may arise from the epithelial-mesenchymal transition (EMT)-associated gene expression reprogramming process
Individual outcomes are not guaranteed and depend on clinical eligibility, biology, and lifestyle factors
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