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Tirzepatide and GIP treatment lead to significantly lower interleukin-1 beta (IL1B) levels and Tirzepatide further lowered interleukin-6 (IL-6) and monocyte-chemoattractant protein-1 (MCP-1) levels, highlighting distinct tissue-specific regulatory roles in pancreatic adipose tissue metabolism and inflammation [32]

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By enforcing lysosomal clearance rather than kinase inhibition, LY-dE#5 potently suppresses EGFR-driven tumor growth and surpasses the efficacy of current tyrosine kinase inhibitors like Osimertinib, highlighting a promising route to overcome therapeutic resistance [65]