"Body protective compound-157 enhances alkali-burn wound healing in vivo and promotes proliferation, migration, and angiogenesis in vitro." Drug Design, Development and Therapy, 2015;9:2485-2499
Previous studies suggested that AFB1/2, AFG1, and AFM1 mainly interacted with HSA binding site I (185), but the binding affinities of the remaining AFs metabolites are not comprehensively investigated in the literature
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2.4 Mitochondrial dysfunction in PD Mitochondrial dysfunction is intricately linked to PD etiology and ferroptosis
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*Genealogy and Human Health*, 2013