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Currently, the medication has not been approved by the FDA and is in phase 3 clinical trials

Therefore, high expression of GLUT1, positively correlate with the proliferative index and equates to the malignant characteristic (Morani et al., 2014)

Takeaway: Its a potential helper, but lifestyle changes (visceral fat loss) are still the heavy lifters

Going back to the IEL cells for a moment, in Crohns disease and ulcerative colitis (UC) these cells can start to attack the gut lining itself, this further creates a cytotoxic environment, which can further exacerbate IELs to produce pro-inflammatory proteins, this can lead to a compromised intestinal lining, along with alterations in the gut microbiome

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[1] This phenomenon appears to be an indirect consequence of the medication's primary therapeutic effects rather than a direct pharmacological action on temperature regulation