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Ferroptosis in organ fibrosis diseases Following acute and chronic injury, the uncontrolled regulation of inflammation in tissues causes excessive ECM deposition and fibrosis, which irreversibly destroys the structure and function of tissues and organs, resulting in organ failure and high mortality [5]
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ROS and reactive nitrogen species can damage biomolecules in the inflammatory milieu, such as DNA, proteins and lipids, and cause malfunction to create a cycle of oxidative stress imbalance that eventually promotes tumorigenesis and progression, including cholangiocarcinoma (90)
Aged animals typically exhibit difficulty with spatial learning tasks, yet Dihexa treatment enabled these older rats to perform at levels comparable to young animals, suggesting the compound's capacity to restore age-compromised cognitive function
This makes it a valuable therapeutic treatment in combating cognitive impairment and various brain disorders
today BPC-157 (Body Protection Compound-157) is a synthetic pentadecapeptide derived from a sequence naturally present in the gastric juices of mammals
