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As different classes of drugs target different aspects of T2DM via distinct mechanisms of action, a combination of treatments is generally more effective at maintaining glycaemic control than monotherapy over the long term [12]
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Our results showed that ethanol exposure for 810 weeks may activate the inflammasome NLRP3, promoting the secretion of the pro-inflammatory factor TNF- in the hippocampus of ethanol-exposed APP/PS1 mice, which is consistent with the results of A depositions and impaired antioxidant capacity
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