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Any disruption to this pathway changes the risk profile of exogenous copper administration
10.1124/jpet.118.247841 J
HCQ modulates SLE-associated atherogenesis through AP-1-mediated T-cell suppression (140) and endothelial stabilization via dual mechanisms: attenuation of TLR signaling and ROS generation, and AMPK-dependent VSMC cycle arrest (140, 141)
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Adenosine modulates hypoxia-induced responses in rat PC12 cells via the A2A receptor J Physiol 1998
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