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Mechanistically, GLP1R is expressed at low levels in the heart and vasculature, raising the possibility that GLP1 might have both direct and indirect actions on the cardiovascular system
Neuroinflammation as a target for treatment of stroke using mesenchymal stem cells and extracellular vesicles
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This cycle continues with the newly formed L-citrulline converting back to L-arginine, creating a sustainable supply of NO precursors
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