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10.1038/mp.2010.136 43 SandinS.LichtensteinP.Kuja-HalkolaR.LarssonH.HultmanC

Incretin-based therapies work either by preventing enzymatic degradation of GLP-1, thus maintaining plasma levels of endogenous GLP-1 (dipeptidyl peptidase 4 inhibitors, DPP4is) or by directly activating GLP-1 receptors and mimicking the action of native GLP-1 (glucagon-like peptide-1 receptor agonists, GLP-1 RAs) [9]

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The brain-heart axis: effects of cardiovascular disease on the CNS and opportunities for central neuromodulation