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B12 deficiency is easily treatable, but the longer you wait, the more difficult recovery becomesand the higher the risk of permanent neurological damage
Furthermore, research continues to explore its impact on brain health
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It is structurally designed to interact with metabolic enzyme pathways and has become a subject of interest in metabolic and mitochondrial research fields
The action of 2-hydroxyphytanoyl-CoA lyase yields formyl-CoA and pristanal
Lifelong treatment may be needed when the body is unlikely to regain the ability to absorb enough vitamin B12, such as autoimmune gastritis or the major surgical causes identified by NICE
