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In a zebrafish model, compared with C3-carnitine, C18-carnitine and C18:1-carnitine can induce the inhibition of OXPHOS, interfere with the energy supply of cardiomyocytes, and lead to a significant impairment of cardiac systolic function, which manifests as a decreased heart rate and reduced shortening of the atrial and ventricular fractions [21]
No, the reported KLOW half life can vary significantly depending on the research model (e.g., in vitro cell culture, in vivo animal models), the route of administration, and even the specific biological matrix being studied
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