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In asthma, the pathogenesis of excessive mucus production is primarily attributed to increased numbers of goblet cells, which are stimulated by inflammatory mediators such as leukotrienes, proteolytic enzymes, and neuropeptides [70, 71]

The results of the DCFH-DA fluorescence probe assay and GSH content assay reflected the same trend, with the addition of metformin reversing the erastin-induced increase in ROS and the decrease in GSH content (Fig

The real mechanism (lowering the stress hormone cortisol that impairs the prefrontal cortex and hippocampus, plus a GABA-mimetic and mild acetylcholinesterase action)

This apparent contradiction may reflect a critical evolutionary adaptation in which cancer cells reprogram lactate metabolism to attain dual benefits (enhanced metabolic flexibility coupled with augmented oxidative stress resistance) thereby facilitating survival under adverse microenvironmental conditions

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This surface-in pattern strongly suggests that a toxin circulates in the CSF of MS patients to induce myelin damage in the adjacent parenchyma