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The present study found increased DNA methyltransferases activity and gene expression of Dnmt1, 3a, 3b, and Tet (2 and 3)
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In addition, intracellular oxidative damage can activate apoptosis via the C-Jun N-terminal kinase (JNK) pathway, a MAPk that activates p53
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Proteomics 5 , 721730 (2008)
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