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The adhesion molecules may initiate the inflammatory process by activating the release of proinflammatory cytokines and chemokines [207]
Special population studies, including evaluation in elderly patients, patients with significant comorbidities, and patients with organ dysfunction, would help establish safety profiles for diverse clinical populations
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Mutations in the potassium voltage-gated channel subfamily q member 4 (KCNQ4) gene have a large contribution to DFNA5 [40], it has also been shown that there is no significant link between DFNA5 and ARHL [41]
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