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Elevated levels of ROS and oxidative damage markers, such as 8-OHdG and MDA, have been detected in the synovial fluid and tissues of RA patients
Chloramphenicol and other drugs having bone marrow suppressant properties may cause a lack of therapeutic response to vitamin B12
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They have an incretin action, reducing glucose plasmatic levels through the stimulation of insulin secretion by the pancreatic beta-cells and the concomitant inhibition of glucagon secretion by alpha-cells
Recent research has shown that cold-stimulated bronchial epithelial cell-derived exosomes (CS-BECs-exo) exacerbate sepsis-induced ALI by increasing mitochondrial ROS via the HMGB1/RAGE/Nrf2/HO-1 pathway, suggesting a novel extracellular vesicle-mediated mechanism of mitochondrial oxidative stress in ARDS (Wang et al., 2025)
Wald NJ, Watt HC, Law MR, Weir DG, McPartlin J, and Scott JM (1998)
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