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DTP cells survive under chemotherapeutic pressure by actively engaging nongenetic adaptive mechanisms, such as CYP3A-mediated drug detoxification, thereby driving adaptive resistance and serving as a key cellular source of treatment failure and tumor recurrence in PDAC

The absorption of light by chromophores puts them in a state of high excitation marked by a tendency to undergo rapid chemical reactions with other molecules, including molecular oxygen, which can lead to the production of ROS 3,4,21

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Microarray studies have identified over 4,000 human genes responsive to GHK-Cu at very low concentrations