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In this review, we discuss how iron uptake, regulation and metabolism, including the redistribution of iron in the host defense system during viral infection, can induce ferroptosis

Further research should be performed on personalized Se supplementation approaches using genetics and nanotechnology to optimize the bioavailability and efficacy of the supplements

The fundamental challenge lies in developing pharmacological agents capable of reliably shifting microglial polarization from pro-inflammatory (M1-like) to anti-inflammatory, phagocytic (M2-like) phenotypes, thereby promoting neuroprotection while mitigating disease progression

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Increased GSTP1 expression has been reported in pre-neoplastic lesions in chemically induced animal cancer models (Satoh et al, 1985) and in many human tumours, relative to surrounding normal tissues (Shea et al, 1988)