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Preclinical studies have demonstrated that ambroxol enhanced the antimycobacterial activity of rifampicin against intracellular BCG [27], induced autophagy through the activation of TFEB both in vitro and in vivo models, promoted mycobacterial killing in macrophages
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Our model is complementary to the work of[10] since it focuses on the detailed biochemical mechanisms by which of APAP is detoxified in the liver under both normal and overdose situations
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