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Thus, the neuroprotective effects exhibited by curcumin in preclinical TBI models can be attributed to its ability to modulate oxidative stress, neuroinflammation, mitochondrial dysfunction, and autophagy
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10.1016/j.regpep.2009.11.005 20 GojkovicS.KrezicI.VrdoljakB.MalekinusicD.BarisicI.PetrovicA.et al (2020)
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If currently on cagrilintide 2.4 mg plus semaglutide 2.4 mg, you would maintain the cagrilintide dose and replace semaglutide with retatrutide at a starting dose of 1-2 mg, then titrate up
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