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The relationship between GLP-1 and insulin appears to be bidirectional, as insulin stimulates GLP-1 secretion from the enteroendocrine cells and insulin resistance, in vitro and in vivo, is associated with impaired GLP-1 secretion 73

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Studies have demonstrated that localized accumulation of EAT surrounding the atriarather than overall cardiac adiposityplays a pivotal role in inducing conduction abnormalities that lead to AF (108)

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