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10.1016/S0891-5849(02)00884-5 59 CuiZ.LockmanP
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This investigation builds on substantial evidence linking mitochondrial metabolism to cytokine regulation and frames SLC25A40 as a putative myeloid-relevant controller of mtGSH 1,5
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Thus, observed potent induction of ferroptosis, GPX4-dependent novel suppression of mTOR pathway and DNA damage repair response in preclinical in vitro model of TC supports GPX4 targeting for therapeutic benefit in advanced therapy-resistant thyroid cancers
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