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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis
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Improved Joint Mobility : Enhances flexibility and movement
Regulates inflammation Helping the body balance its immune response and reduce chronic inflammation
*JAMA Internal Medicine*, 173(5), 386-388
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