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Since fibrosis is the most compelling predictor of outcomes, current and future therapies must focus on improving fibrosis by indirectly reducing pro-fibrotic inflammatory signaling or directly abrogating fibrosis
Sustainable progress matters: The goal is not quick, temporary results
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[11] [1] In some participants, liver fat was reduced to below the 5% threshold conventionally used to define hepatic steatosis, suggesting potential resolution of fatty liver in those individuals
They reduce appetite from different receptor systems, which is precisely why an amylin analog is studied alongside a GLP-1 analog in CagriSema rather than as a substitute for one
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