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Sustained inhibition of NPY/AgRP neuronal activity by FGF1
The detrimental effects of ROS on cartilage homeostasis can be effectively alleviated by augmenting cellular antioxidant activity under stress conditions, and several attempts have been made to treat OA by targeting the regulators involved in oxidative stress production in cartilage 84,85,86
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Whats more, it inhibits ECM deposition, alleviates glomerular basement membrane thickening, and mitigates renal tubular atrophy, thus attenuating various histopathological injuries associated with diabetes (Sun et al., 2015)
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