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The brain pathology of this disease is similar to that seen in oxidative damage resulting from sulfhydryl depletion due to chronic mercury poisoning (Minamata disease) and is also compatible with decreased cerebral glutathione levels (09)
As side effects diminish and weight loss benefits accumulate, the indirect risk factors that drive UTI vulnerability decrease while protective factors increase
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The enhanced mitochondrial oxidative stress leads to DA oxidation with generation of DAQs, conjugation of DAQs with GBA1, inhibition of GBA1 enzymatic activity, lysosomal dysfunction, and accumulation of deleterious -syn protein [10]
10.7554/eLife.56749 82 JinJ.ByunJ
The motor nerve conduction study also confirmed the absence of unmyelinated processes in the tail caudal nerves following surgical spinal cord injury
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