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Figure 1 2.2 CytokineNEU interactions drive inflammatory activity Matrix metalloproteinases (MMPs), a family of zinc-requiring endopeptidases, play critical roles in extracellular matrix degradation and tissue remodeling, with their overexpression implicated in immune-mediated tissue damage ( 2.3 Microbiome-NEUs crosstalk NEUs play a paradoxical role in intestinal pathology, contributing to both inflammatory responses and tissue protection ( + neutrophils generate elevated ROS and antimicrobial peptides, strengthening mucosal defense while suppressing pro-inflammatory cytokine expression ( + neutrophils produce IL-22, a key mediator in maintaining epithelial homeostasis ( 2.4 Dynamic behavior of NEUs NEUs occupy diverse functional states along a continuum from immune-enhancing/pro-resolving phenotypes to dysfunctional, hyperinflammatory programs often described as exhausted ( 3 Inhibition of NEU in UC 3.1 Inhibition of NEU-derived ROS and pro-inflammatory cytokines Hesperidin methyl chalcone (HMC), a citrus flavonoid derivative, exerts antioxidative, anti-inflammatory, and analgesic effects by enhancing colonic glutathione levels and antioxidant capacity, thereby limiting NEU infiltration and mucosal damage in UC (65)

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