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Pro-inflammatory and proatherogenic mCRP, but not nCRP, induces ROS monocyte/macrophage production and facilitates macrophage uptake of necrotic cells (170, 192) contributing to foam cell formation, atherosclerotic plaque formation and plaque rupture or destabilization (190, 191)

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This is especially crucial for patients with neurological symptoms, which can easily become permanent if under-treated long enough

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DNA damage induced by endogenous aldehydes: current state of knowledge

2014 Dec;102(6):1518-27 (Ver) Mora-Esteves C, Shin D