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For a 100kg individual, that translates to 22.5kg lost versus 2.4kg
Unsurprisingly, research indicates that patients with FMS exhibit both of the above defects in central pain processing: Chemical mediators involved in pronociception have been shown to be abnormal in FMS
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New insights into the pathogenetic mechanisms implicate endothelial dysfunction, immune complex deposition and the interplay of pro-inflammatory cytokines with prothrombotic factors, which collectively promote thrombus formation
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