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1 Introduction Sepsis-associated encephalopathy (SAE) represents a severe complication of sepsis, defined by diffuse brain dysfunction without direct central nervous system (CNS) infection ( Oxidative stress is central to SAE pathogenesis, driven by an imbalance between excessive reactive oxygen species (ROS) production and impaired antioxidant defense systems ( The antioxidant defense system, especially GSH metabolism, is linked to neurodegenerative diseases ( Proteomics and metabolomics enable systematic profiling of protein expression and metabolic signatures

Selon une tude, les doses orales courantes (jusqu 2000 g/jour) sont sans danger

Immunity 14 , 617629 (2001)

Discovery of SLC16A9 and SLC22A1 as regulators of acylcarnitines associated with Alzheimers disease