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(2) a second, VEGF-independent Srccaveolin-1eNOS angiogenesis pathway that operates even in low-VEGF environments

(3) Thus, GHK-Cu is posited to act as a repair signal, capable of interacting with enzymes, ion channels, and cell-surface receptors, with reported downstream potential on gene expression

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It also stimulates focal adhesion kinase (FAK)-paxillin complexes, which help cells migrate and attach during tissue reconstruction