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When disc material herniates, it triggers an inflammatory cascade involving cytokines, prostaglandins, and other chemical mediators

As evident from the above discussions, researchers have mostly correlated the cellular radiosensitivity with the GPx activity level which is majorly contributed by GPx1 (because of its higher abundance) [1, 8, 9]

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The acetyl bond is placed on its thiol group or sulfur group, which prevents oxidation and allows the molecule to pass diffusively into the cell after absorption in the gut