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GLP-1s peripheral effects include insulin secretion, gut emptying, and inhibition of glucagon secretion
Key mechanisms include HMGB1/ACSL4 pathway, the hypoxia/HIF1-SUMO axis, and GPX4 degradation
Its deleterious effects are primarily mediated through oxidative stress, neuroinflammatory responses, and apoptotic pathways
Frequent monitoring of trough blood levels of cyclosporine and tacrolimus is recommended and adjust the dose when appropriate [see Clinical Pharmacology ( 12.3)]
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