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Moreover, microbial transplantation studies in germ-free mice with human commensals containing a functional (but not a deletion mutant) microbial cutC gene (responsible for cholineTMA transformation) (23) and genetic manipulation (both gain of function and loss of function) of host hepatic flavin monooxygenase 3 have been shown to modulate host TMAO levels, platelet responsiveness, and thrombosis potential in vivo (24)
Considering the potential appetite-reducing effects of GLP-1 RAs, diet-restricted control groups were included for both drugs, ensuring consistent food intake between treatment and control groups, thereby minimizing any dietary bias
( Cutibacterium acnes , were looking at you.) (11 & 12) There are also a few other things that come into play here, too
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