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Interventions in this direction mainly manifest in two complementary logics: one is to indirectly block hypoxia-induced pro-oxidant signaling axes by inhibiting HIF function [486]

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Our study found that the GSH-triggered long-distance calcium-mediated defense response was suppressed in GCaMP3/glr3.3 but nearly unaffected in GCaMP3/glr3.6 (Fig