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Because the loss of these neurotrophic factors has been implicated in the pathogenesis of various neurodegenerative diseases, our results provide a potentially important mechanism whereby Hex may ameliorate or delay neurodegeneration in AD

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Surprisingly, CTLA-4 blockade has been observed to increase the expression and activity of the enzyme indolamine 2,3-dioxygenase (IDO), which could further contribute to immune exhaustion (234)