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Oxidative stress contributes to PD pathogenesis through multiple mechanisms, including mitochondrial dysfunction, protein misfolding, and neuroinflammation [159]

Many tumor cells are highly dependent on glutamine to supplement and renew the TCA cycle (Yang et al., 2017)

Hair repigmentation associated with the use of brentuximab

Experiencing a persistent metallic taste can be an unexpected and distressing symptom

Chronic tendinopathy differs from acute tears in one key way: the tissue is already remodeling in a disorganized direction

CD38 inhibition by apigenin ameliorates mitochondrial oxidative stress through restoration of the intracellular NAD+/NADH ratio and Sirt3 activity in renal tubular cells in diabetic rats