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Here we show, by studying mice with B cell-specific ablation of the catalytic subunit of glutamate cysteine ligase ( Gclc ), that glutathione synthesis affects homeostasis and differentiation of MZB to a larger extent than FoB, while glutathione-dependent redox control contributes to the metabolic dependencies of FoB
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Studies have shown that various pancreatic cancer cell lines preferentially degrade glutamine to provide nitrogen for polyamine synthesis
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and regulates the mechanisms essential to homoeostasis, which includes enzymatic activity, protein synthesis, gene expression, and mitochondrial integrity
